cardio:arrhythmias:bradycardia
Bradycardia Algorithm
Bradycardia is defined as:
- Heart rate < 60 bpm
Clinical relevance depends on:
- Symptoms
- Hemodynamic stability
- Presence of AV block
This page follows the Dysrhythmias Module and mirrors ACLS principles.
Step 1 – Identify Symptomatic vs Asymptomatic
Assess:
- Hypotension
- Altered mental status
- Signs of shock
- Ischemic chest discomfort
- Acute heart failure
If NO symptoms:
→ Monitor → Evaluate underlying cause → No emergent therapy required
If SYMPTOMATIC:
→ Proceed to Step 2
Step 2 – Determine Rhythm
Common causes:
- Medication-induced bradycardia
Step 3 – Immediate Treatment (Unstable Bradycardia)
First-Line Medication
- 1 mg IV push
- Repeat every 3–5 minutes
- Maximum total dose: 3 mg
Mechanism:
- Blocks vagal tone at SA/AV node
Not effective in:
- Infranodal block
- High-grade distal AV block
Step 4 – If Atropine Ineffective
Escalate immediately:
Transcutaneous Pacing
OR Start Infusion
Special Scenarios
High-Grade AV Block (Mobitz II or Complete Heart Block)
Beta-Blocker Toxicity
Calcium Channel Blocker Toxicity
Digoxin Toxicity
- Consider Digoxin Immune Fab
Full Flow (Quick View)
Bradycardia → Is patient symptomatic?
NO → Monitor
YES → Give Atropine
If no response → Transcutaneous pacing
If pacing unavailable → Dopamine or Epinephrine infusion
Consider reversible causes.
Reversible Causes
- Hypoxia
- Electrolyte abnormalities
- Drug effects
- Increased vagal tone
- Ischemia
Always treat underlying cause.
Clinical Pearls
- Treat the patient, not just the number.
- Asymptomatic bradycardia does not require emergent therapy.
- Atropine works at the AV node — not distal conduction tissue.
- Early pacing saves time in high-grade block.
- Always review medication list.
Related
cardio/arrhythmias/bradycardia.txt · Last modified: by andrew2393cns
