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--- office_hours:inflammation:start [2026/02/15 17:50] – [High-Yield Framework] andrew2393cns
+++ office_hours:inflammation:start [2026/02/15 17:52] (current) – [High-Yield Framework — How Immune Drugs Differ] andrew2393cns
@@ Line 114: / Line 114: @@
 ^ Depth of Action ^ What The Drug Is Really Doing ^ Drug Classes ^ Typical Clinical Use ^
 | Surface (symptom relief) | Blocks mediators already released | [[allergy:drug_classes:first_generation_h1|Antihistamines]], [[immunology:biologics:anti_ige|Omalizumab]] | Seasonal allergies, urticaria |
-| Local chemical inflammation | Blocks inflammatory chemicals (prostaglandins / leukotrienes) | [[eicosanoids:nsaids:start|NSAIDs]], [[cardio:antiplatelet:aspirin|Aspirin]], [[respiratory:leukotriene_modifiers:5lo|Zileuton]], [[respiratory:leukotriene_modifiers:ltra|Montelukast]] | Pain, fever, mild asthma |
+| Local chemical inflammation | Blocks inflammatory chemicals (prostaglandins / leukotrienes) | [[eicosanoids:nsaids:start|NSAIDs]], [[cardio:antiplatelet:aspirin|Aspirin]], [[respiratory:drugs:zileuton|Zileuton]], [[respiratory:drugs:montelukast|Montelukast]] | Pain, fever, mild asthma |
-| Broad immune suppression | Turns off inflammatory gene production | [[immunology:corticosteroids:start|Corticosteroids]] | Severe inflammation, flares |
+| Broad immune suppression | Turns off inflammatory gene production | [[endocrine:drug_classes:corticosteroids|Corticosteroids]] | Severe inflammation, flares |
 | Targeted immune modulation | Blocks specific immune communication pathways | [[immunology:biologics:anti_tnf_agents|TNF inhibitors]], [[immunology:biologics:start|IL inhibitors]], [[immunology:jak_inhibitors:start|JAK inhibitors]] | Autoimmune disease, severe asthma, IBD |
 </WRAP>